SERVICES | DIAGNOSIS

PRE-TEST: ACTUAL testing for Lyme disease and co-infections1. Stage: Immunological function tests-LTT (provides evidence as early as 14 days after the bite or can indicate "reactivation" or inadequate therapy 2nd stage: ELISA test (IgM and IgG Ab) Western blot (pos. only after 3rd stage). -4 weeks post infectionem), unfortunately not very sensitive!T-cellspot® Borrelia as a therapy success control and to differentiate between fresh and chronic infectionDiagnostics of chronic Lyme disease optionally also using CD 57 or "shared epitope" testing" to determine a genetic predisposition to the development of Lyme borreliosis increased IFN-α activity, IL-23, 10 & TH 17 values in typical clinic direct detection of the pathogen DNA using a PCR in the tick found (tick PCR) or in various. human sample materials (such as urine, blood, cerebrospinal fluid, synovial fluid, skin biopsies); the PCR test of urine is the most suitable

TBE-specific IgM and IgG Abs in the serumAnaplasmosis & Babesiosis: IFT (immunofluorescence Ab detection for IgM and IgG Abs) - "Seronegative Lyme borreliosis with corresponding symptoms or the failure of a Borrelia-specific therapy can be achieved through a Anaplasmosis or babesiosis can be explained!"EBV serology: EBV-VCA-IgG (including avidity/RAI), -IgM, EA-IgG, EBNA-1-IgG, VCA p22, T-cellspot EBVother: Rickettsia, Coxiella, Branhamella , Mycoplasma, CMV, Coxsackien, HSV, ...

Among other things, there are rare but noticeable bite marks on the scalp "Scalp eschar" (also called TIBOLA / SENLAT) which are associated with lymphadenopathy in the neck and can be caused by either Rickettsia raoultii or B. afzelii. Accompanying symptoms include fever, fatigue and skin rashes.

Recognize Alpha-Gal Syndrome! This allergy to the sugar galactose-alpha-1,3-galactose (alpha-gal) is caused in some people by the bite of certain species of ticks (B. lonestarii) and manifests itself, among other things, in excruciating itching (urticaria).

Morgellons disease - MD, was only described in more detail in 2011 by Peter Mayne et al as a skin disease associated with Lyme disease (B. burgdorferi) and is to be understood as a dermal manifestation of the multisystem disease Lyme disease - "Borrelial dermatitis". Clinically, MD is characterized by general unexplained itching, nodular foci of inflammation, severely itchy poplars, hypo-hyperpigmentation, crawling and stinging skin sensations, etc. Morgellons appear at 60x magnification as different colored keratin or collagen-containing filaments that can be found in skin lesions or can be seen under intact skin at high magnification. How they arise in the skin and what role Borrelia play in this has now been discovered by the microbiologist Marianne Middelveen et al. and Dr. Raphael Stricker (doctor, researcher and Borrelia specialist) researched it in detail in 2011 and is morphologically very similar to digital dermatitis, or DD for short, in cattle and elk. I consider labeling patients with MD as psychotic sufferers (as "delusional disorders" (ICD-10: F 22.0; DSM-IV 297.1/Dermatozoenwahn (DW))) unacceptable and hostile to patients from a professional and ethical perspective due to the clear study situation if psychiatric comorbidities are clearly more common in patients with Lyme disease/LD and MD due to the high level of suffering. (https://doi.org/10.12688/f1000research.2-118.v1)

Using special laboratory tests, I find out in my patients with a more severe clinical course which Borrelia strains or their numerous sub-forms (ssp./strains) such as B. burgdorferi, garinii, afzelii, japonica, vincentii, ( approx. 25 more)... .or which co-infections are present. In addition to the regular antibody test in the serum, the rel. to highlight the new Elispot-LTT.

B. miyamotoi (only known in Austria since 2015), hermsii, duttonii, recurrentis and others (approx. 15 species) cause, for example, “endemic, recurrent fever / relapsing fever”.

There are also co-infections such as Neoehrlichia mikurensis, Ehrlichia/Anaplasma phagocytophilum, Rickettsia ssp., Chlamydiae ssp., Francisella tularensis, Bartonella, Coxiella, Babesia, Leptospira, Mycoplasma, Rabies lyssavirus, Toxoplasma, Wolbachia, Yersinia and even viruses such as TBE, EBV, Borna-V., Parvo-V., CMV, herpes simples type I II, HHV6, HIV, among others, have to be detected and treated, which requires very in-depth and complex therapy and further control examinations during the course of the disease .

In some cases, it is not the Borrelia that is responsible for the symptoms, but rather the chlamydia that is present at the same time. (C. pneumoniae, trachomatis) Chlamydia symptoms can look like Alzheimer's disease, multiple sclerosis, fibromyalgia, CFS, myocardial infarctions, insults. Vascular inflammation, arteriosclerosis, prostate hyperplasia/prostatitis, vision problems, migraines and much more, Dr. In this context, Klinghardt speaks of pseudo-MS, pseudo-fibromyalgia, etc.

If symptoms remain after what doctors say is a “successful” antibiotic! On the one hand, these can still be caused by one or more co-pathogens or the Borrelia have left behind permanent tissue damage before they are eliminated, which can continue for months to years = Post-Lyme Syndrome/PLDS! (Examples: permanent cognitive disorders after neuroborreliosis due to the formation of persistent antineural antibodies, dilated cardiomyopathy after Lyme carditis, synovitis after Lyme arthritis, etc.)